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Role of FAS in HIV infection.
Umberto Dianzani
, Thea Bensi
, Andrea Savarino
, Selina Sametti
, Manuela Indelicato
, Riccardo Mesturini
,
Annalisa Chiocchetti
Dipartimento di Scienze della Salute
Risultato della ricerca
:
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›
peer review
Panoramica
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Keyphrases
Apoptosis
100%
HIV Infection
100%
Lymphocytes
66%
Human Immunodeficiency Virus Type 1 (HIV-1)
66%
Genetic Background
66%
Viral
66%
Death Receptor
66%
Spontaneous Apoptosis
66%
AIDS/HIV
33%
Drug Use
33%
Cytopathic Effect
33%
HIV Patients
33%
Gp120
33%
Infected Cells
33%
CD4+ T
33%
Highly Active Antiretroviral Therapy (HAART)
33%
Indirect Mechanism
33%
TNF Receptor 1 (TNFR1)
33%
Survival Factor
33%
Apoptosis Resistance
33%
T Cell Apoptosis
33%
Stem Cell Exhaustion
33%
Intrinsic Resistance
33%
Uninfected Cells
33%
Uncontrolled Infection
33%
HIV-infected Cells
33%
TRAIL Receptors
33%
HIV-1 Protease
33%
CD4+ T Cell Depletion
33%
Biological Differences
33%
Clinical Differences
33%
Immunology and Microbiology
Human Immunodeficiency Virus Infection
100%
Programmed Cell Death
100%
Human Immunodeficiency Virus
71%
T-Helper Cell
28%
Human Immunodeficiency Virus 1
28%
Death Receptor
28%
Genetic Background
28%
Lymphocyte
28%
Acquired Immune Deficiency Syndrome
14%
T Cell
14%
Cytokine
14%
Tumor Necrosis Factor
14%
Cytopathic Effect
14%
Survival Factor
14%
T Cell Depletion
14%
TNF related apoptosis inducing ligand
14%
Vpr
14%