Effects of Pin1 loss in Hdh^Q111 knock-in mice

Neuroscience

• Huntington's Disease 100%
• Huntingtin 100%
• In Vitro 33%
• In Vivo 33%
• Neurodegenerative Disorder 33%
• Neurodegeneration 33%
• P53 33%
• DNA Damage Response 33%
• Gliosis 33%
• Coding Region 33%
• CAG Repeat 33%
• Isomerase 33%
• Proline Derivative 33%
• Catenin 33%
• Programmed Cell Death 33%

Biochemistry, Genetics and Molecular Biology

• Huntingtin 100%
• P53 33%
• Knockout Mouse 33%
• Coding Region 33%
• Catenin 33%
• Next Generation Sequencing 33%
• DNA Damage Response 33%
• Genetic Ablation 33%
• CAG Repeat 33%
• Prolyl Isomerase 33%
• Gliosis 33%
• Breed 33%
• Programmed Cell Death 33%

Keyphrases

• Knock-in Mouse Model 100%
• Pin1 100%
• Huntington's Disease 33%
• Mutant Huntingtin 33%
• Neurodegenerative Diseases 11%
• Coding Region 11%
• Aging 11%
• Catenin 11%
• Neurodegeneration 11%
• Early Events 11%
• DNA Damage Response 11%
• Gliosis 11%
• Disease Pathogenesis 11%
• Genetic Ablation 11%
• CAG Repeat 11%
• Prolyl Isomerase Pin1 11%
• P53-dependent Apoptosis 11%
• Next-generation Sequencing Technology 11%
• HTT Gene 11%
• Nuclear Inclusions 11%
• Precise Model 11%