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Downregulation of E2F1 during ER stress is required to induce apoptosis
Vittoria Pagliarini
, Paola Giglio
, Paolo Bernardoni
, Daniela De Zio
, Gian Maria Fimia
, Mauro Piacentini
,
Marco Corazzari
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Keyphrases
Induced Apoptosis
100%
Endoplasmic Reticulum Stress (ER stress)
100%
E2F Transcription Factor 1 (E2F1)
100%
Endoplasmic Reticulum
22%
Unfolded Protein Response
22%
Inositol-requiring Enzyme 1 (IRE1)
22%
Activating Transcription Factor 6 (ATF6)
22%
Tumor
11%
Apoptosis
11%
Tumor Cells
11%
DNA Damage
11%
Cell Death
11%
Cell Survival
11%
Chemoresistant
11%
Pro-apoptotic Factors
11%
Knockdown
11%
Pro-apoptotic
11%
Late Events
11%
Transformed Cells
11%
Anticancer Treatment
11%
Noxa
11%
Apoptosis Induction
11%
Stress-driven
11%
Execution Phase
11%
X-box Binding Protein 1 (XBP1)
11%
Potential Therapeutic Strategies
11%
Stress Dependence
11%
Endoplasmic Reticulum Stress-induced Apoptosis
11%
Pma1
11%
E2F7
11%
ERN1
11%
Puma
11%
BBC3
11%
Biochemistry, Genetics and Molecular Biology
Downregulation
100%
Endoplasmic Reticulum Stress
100%
E2F1
100%
Programmed Cell Death
100%
Unfolded Protein Response
22%
ATF6
22%
Promoter Region
11%
Cell Death
11%
Upregulation
11%
Cell Survival
11%
Anticancer
11%
Puma
11%
Medicine and Dentistry
Programmed Cell Death
100%
Downregulation
100%
Endoplasmic Reticulum Stress
100%
Endoplasmic reticulum
28%
Unfolded Protein Response
28%
Cell Death
14%
Tumor Cell
14%
Cell Survival
14%
Promoter Region
14%
Upregulation
14%
Toxic Substance
14%
Transformed Cell
14%
Tumor
14%
Immunology and Microbiology
Downregulation
100%
Endoplasmic Reticulum Stress
100%
Programmed Cell Death
100%
Endoplasmic reticulum
28%
Unfolded Protein Response
28%
Promoter Region
14%
Upregulation
14%
Tumor Cell
14%
Cell Survival
14%
Cell Death
14%
Transformed Cell
14%
Puma
14%