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Continuous petri nets and microrna analysis in Melanoma
Giulia Russo
,
Marzio Pennisi
, Roberta Boscarino
, Francesco Pappalardo
Risultato della ricerca
:
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›
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Keyphrases
Melanoma
100%
MicroRNA
100%
BRAF Inhibitor (BRAFi)
100%
Petri Net Analysis
100%
MicroRNA Analysis
100%
Hsa-miR-132
100%
P120RasGAP
100%
Continuous Petri Nets
100%
Cancer Therapy
50%
Melanoma Cancer
50%
High Concentration
50%
Expression Level
50%
Treatment Strategy
50%
Clinical Outcomes
50%
Targeted Therapy
50%
Mitogen-activated Protein Kinase
50%
Phosphoinositide 3-kinase (PI3K)
50%
Aggressive Tumor
50%
Possible Treatments
50%
Signaling Cascades
50%
Akt Activation
50%
Petri Net Model
50%
Antisense Oligonucleotides
50%
Selective Inhibitor
50%
AKT1
50%
Treatment Interventions
50%
Pathway Activation
50%
Melanoma Patients
50%
PDK1
50%
Inhibitor Drug
50%
Kinase Cascade
50%
BRAF V600E
50%
Melanoma Development
50%
Melanomagenesis
50%
Crucial Nodes
50%
Common Data
50%
Cancer Genomic Data
50%
Genomic Data Commons
50%
Biochemistry, Genetics and Molecular Biology
Mitogen-Activated Protein Kinase
100%
MicroRNA
100%
Protein Kinase B
100%
MiR-132
100%
Kinase
50%
Phosphotransferase
50%
Dynamics
50%
Oligonucleotide
50%
Antisense
50%
Phosphoinositide-Dependent Kinase-1
50%
Genomics
50%
Cancer Genomics
50%
Medicine and Dentistry
microRNA
100%
Melanoma
100%
Malignant Neoplasm
50%
Mitogen-Activated Protein Kinase
50%
Protein Kinase B
50%
microRNA 132
50%
Neoplasm
25%
Antisense Oligonucleotide
25%
Cancer Therapy
25%
Phosphotransferase
25%
Phosphoinositide Dependent Protein Kinase 1
25%
Pharmacology, Toxicology and Pharmaceutical Science
Melanoma
100%
microRNA
100%
Malignant Neoplasm
75%
Protein Kinase B
50%
microRNA 132
50%
Neoplasm
25%
Antisense Oligonucleotide
25%
Phosphotransferase
25%
Phosphoinositide Dependent Protein Kinase 1
25%