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The IL-17F/IL-17RC Axis Promotes Respiratory Allergy in the Proximal Airways

  • Antonella De Luca
  • , Marilena Pariano
  • , Barbara Cellini
  • , Claudio Costantini
  • , Valeria Rachela Villella
  • , Shyam Sushama Jose
  • , Melissa Palmieri
  • , Monica Borghi
  • , Claudia Galosi
  • , Giuseppe Paolicelli
  • , Luigi Maiuri
  • , Jan Fric
  • , Teresa Zelante

Research output: Contribution to journalArticlepeer-review

Abstract

The interleukin 17 (IL-17) cytokine and receptor family is central to antimicrobial resistance and inflammation in the lung. Mice lacking IL-17A, IL-17F, or the IL-17RA subunit were compared with wild-type mice for susceptibility to airway inflammation in models of infection and allergy. Signaling through IL-17RA was required for efficient microbial clearance and prevention of allergy; in the absence of IL-17RA, signaling through IL-17RC on epithelial cells, predominantly by IL-17F, significantly exacerbated lower airway Aspergillus or Pseudomonas infection and allergic airway inflammation. In contrast, following infection with the upper respiratory pathogen Staphylococcus aureus, the IL-17F/IL-17RC axis mediated protection. Thus, IL-17A and IL-17F exert distinct biological effects during pulmonary infection; the IL-17F/IL-17RC signaling axis has the potential to significantly worsen pathogen-associated inflammation of the lower respiratory tract in particular, and should be investigated further as a therapeutic target for treating pathological inflammation in the lung.

Original languageEnglish
Pages (from-to)1667-1680
Number of pages14
JournalCell Reports
Volume20
Issue number7
DOIs
Publication statusPublished - 15 Aug 2017
Externally publishedYes

Keywords

  • ABPA
  • IL-17F/IL-17RC axis
  • Th17 immunity
  • allergy
  • respiratory infections

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